Related Questions

Digoxin toxicity includes
A Altered color vision
B Bradycardia
C Ventricular tachycardia
D Constipation
E Atrial fibrillation
Digoxin is likely to be useful in the treatment of
A Thyrotoxic atrial fibrilation
B Ventricular tachycardia
C Atrial flutter
D Hypertrophic obstructive cardiomyopathy
E Re-entrant tachycardia in Woll Parkinson-White Syndrome
The beneficial effects of digoxin in congestive heart failure are
A Increased force of contraction
B Slowing of A V nodal conduction
C Reduction in preload
D Reduction in afiterload
E Increased myocardial automaticity
Digoxin acts by
A Inhibition of Na-K ATPase pump
B Sensitization of baroreceptors
C Activation of sodium calcium exchange mechanism
D Decreases parasympathetic activity'
E Increases sympathetic activity
Digoxin toxicities are manifested as
A Vomiting
B Bradycardia
C Ventricular tachycardia
D Headache
E Hypertension
Clinical toxicity occurs in all of the following micro nutrients except :
A Fluoride
B Copper
C Iron
D Iodine
Following are the signs of O2 toxicity
A Seizure
B Retrolental fibroplasia
C Dry cough
D Loss of deep tendon reflex
E Palpitation
Haemodialysis has an important role in the treatment of toxicity of
A Ethylene glycol
B Digoxin
C Amiodarone
D Metformin
E Paracetamol
Toxicity of cyclosporine are
A Hyperkalaemia
B Hypertension
C Nephrotoxicity
D Poor wound healing
E Hepatitis
Toxicity of a drug includes
A Bleeding from heparin overdose
B c. Hypotension from prazosin administration
C Hepatocellular damage from paracetamol overdose
D Renal tubular necrosis from gentamicin administration
E Severe bronchoconstriction from penicillin administration

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