Related Questions

Congenital adrenal hyperplasia is associated with
A Autosomal recessive pattern of inheritance
B Precocious puberty
C A salt losing syndrome
D 21 beta-hydroxylase deficiency
E Male pseudohermaphroditism
Hyperplasia
A Results in increased cell numbers
B Occurs in uterus
C Results in increased cell size
D Occurs in myocardium
E Occurs in brain tissue
Hyperplasia is
A Seen in organs made of labile cells
B A reversible adaptive change
C Lead to malignancy in some cases
D Never associated with hypertrophy
E Seen in epithelium only
Pathologic hyperplasia associated with an increased risk of malignancy includes
A Endometrial hyperplasia
B Atypical ductal hyperplasia of breast
C Nodular prostatic hyperplasia
D Hyperplasia of gastric pits
E Chief cell hyperplasia
In benign prostatic hyperplasia
A IPSS score is the single most important tool used in the evaluation of patients with BPH
B TUR syndrome results from a hypervolumic
C hyponatraemic state
D On digital rectal examination prostatic size correlates poorly with symptoms
E After TURP
F prostatic carcinoma will not occur
G PSA is only prognostic factor in BPH management
Endometrial hyperplasia is associated with
A Anovulatory cycles
B PCOS
C Hypothyroidism
D Combined oral pill
E Hyperprolactinemia
The net effects of stimulation of baroreceptors are
A Venodilation of peripheral circulatory system
B To decrease heart rate
C To decrease arterial blood pressure
D Venodilation of pulmonary vessels
E To increase force of contraction of cardiac muscle
Vagal stimulation causes
A Fall in blood pressure
B Decreased cardiac output
C Constriction of pupil
D Relaxation of bronchial smooth muscles
E Decreased intestinal secretion
Duration of inspiration is decreased and rate of ventilation is increased due to stimulation of
A Pneumotaxic center
B Apneustic center
C Vagus nerve
D Nucleus of tractus solitarius
E Ventral respiratory group
The postsynaptic neuron becomes fatigued after repeatative stimulation of an excitatory synapse at a rapid rate. This is due to
A Exhaustion of neurotransmitter store in presynaptic terminal
B Progressive inactivation of many postsynaptic receptors
C Slow development of abnormal concentration of ions in postsynaptic neurons
D Decrement of number of calcium channels of presynaptic terminal
E Increment of cholinesterase in synaptic cleft

Next steps