Diabetes mellitus MCQ 22+

Test your knowledge with important Diabetes mellitus MCQ and their applications. These MCQs are beneficial for competitive exams too. Explore 52+ more Diabetes mellitus MCQs on Bissoy. Bissoy App

• Musculo-skeletal complications of diabetes mellitus include
  ক

  Neuropathic joint

  খ

  Adhesive capsulitis of shoulder joint

  গ

  Diffuse idiopathic skeletal hyperostosis

  ঘ

  Osteoporosis

  ঙ

  Osteopetrosis
• Statement about hypoglycaemic agents include
  ক

  Glipizide produces less hypoglycemia compared to Glibenclamide

  খ

  Pioglitazone may provoke cardiac congestion

  গ

  Metformin increases hepatic glucose out put

  ঘ

  Repeglinide is euglycemic

  ঙ

  Insulin glergine is shorter acting than isophene insulin
• Macrovascular complicatons of Diabetes mellitus are
  ক

  Cerebrovascular disease

  খ

  Peripheral arterial disease

  গ

  Retinopathy

  ঘ

  Neuropathy

  ঙ

  Nephropathy
• Regarding treatment of diabetes mellitus
  ক

  Pioglitazone acts as an insulin sensitizer

  খ

  Peptidyl peptidase-IV inhibitor acts as insulin secretogogue

  গ

  Dosage of insulin should be increased in patient with somogyi effect

  ঘ

  Two insulin sensitizer cannot be given

  ঙ

  Metformin cannot be given in non-obese patients
• In patients with diabetes mellitus
  ক

  Blood pressure control target should be lower than that of non diabetic

  খ

  Lipid control target should be lower than that of non-diabetic

  গ

  Periodic fundoscopic examination reduces eye complications

  ঘ

  Angiotensin converting enzyme inhibitor do not prevent progression of nephropathy

  ঙ

  Good glycaemic control do not prevent major diabetic complication
• Secondary diabetes mellitus is associated with
  ক

  Haemochromatosis

  খ

  Acromegaly

  গ

  Pancreatic carcinoma

  ঘ

  Frusemide

  ঙ

  Hypothyroidsm
• The following statements about Pharmacotherapy of Diabetes Mellitus are correct
  ক

  Insulin therapy is always associated with some degree of hypoglycaemia

  খ

  Resistance against human insulin is best treated by changing to bovine insulin

  গ

  Pregnant diabetic patients are best treated by Biguanides because they are not teratogenic

  ঘ

  Human insulin is best therapeutically because of lesser chance of hypoglycaemia

  ঙ

  α-glucosidase inhibitor therapy is equally effective as Sulphonylurea drug in NIDDM
• Skin manifestations in diabetes mellitus are
  ক

  Necrobiosis lipoidica

  খ

  Shin spot

  গ

  Xerosis

  ঘ

  Spider angioma

  ঙ

  Hypertrichosis
• Gestational diabetes mellitus are associated with
  ক

  Intra uterine growth retardation

  খ

  Big baby

  গ

  Pre eclampsia

  ঘ

  Hyperemesis gravidarum

  ঙ

  Polyhydramnios
• Diabetic dermopathy includes
  ক

  Bulla

  খ

  Hyperpigmentation

  গ

  Yellow discoloration of skin

  ঘ

  d Necrobiosis lipoidica

  ঙ

  Ichthyosis
• In diabetic ketoacidosis there is
  ক

  Decreased breakdown of glucose

  খ

  High anion gap acidosis

  গ

  Decreased breakdown of ketones

  ঘ

  Decreased breakdown of glycogen

  ঙ

  Decreased breakdown of fat
• In management of diabetes mellitus
  ক

  Structured education program is mandatory

  খ

  Many oral hypoglycemic agents cause weight gain

  গ

  Basal bolus insulin regime has less chance of hypoglycemia [Davidson’s/750]

  ঘ

  Metformin impairs renal functions

  ঙ

  Incretin drugs cannot be combined with other oral drugs
• In diabetic ketosis, there is decreased breakdown of
  ক

  Glucose

  খ

  Ketones

  গ

  Glycogen

  ঘ

  Fat

  ঙ

  Amino acid
• Type 1 diabetes mellitus
  ক

  Shows seasonal incidence

  খ

  Always requires insulin

  গ

  Has strong familial predisposition

  ঘ

  Is characterized by insulin resistance

  ঙ

  Shows high level of C- peptide
• Severe uncontrolled diabetes mellitus leads to a raised
  ক

  H+ ion concentration in body fluid

  খ

  Plasma K+ ion concentration

  গ

  Urinary specific gravity and osmolality

  ঘ

  Blood volume

  ঙ

  Arterial PCO2
• Risk factors for diabetic kidney disease are
  ক

  Poor glycemic control

  খ

  Presence of microvascular complication

  গ

  Preexisting hypertension

  ঘ

  High protein diet

  ঙ

  Obesity
• Diagnostic criteria for diabetes mellitus are
  ক

  HbA1C≥6.5%

  খ

  2-hour plasma glucose ≥11.1 mmol/L during oral glucose tolerance test

  গ

  Fasting plasma glucose ≥ 7.8 mmol/L

  ঘ

  Presence of glucose in the urine

  ঙ

  Presence of ketone bodies in the urine
• Hyperosmolar non-ketotic coma is characterized by
  ক

  Severe dehydration

  খ

  Very high plasma glucose level

  গ

  Moderate to severe acidosis

  ঘ

  Hyperventilation

  ঙ

  Ketonemia
• Hyperglycemic hyperosmolar state is characterized by
  ক

  Hyperglycemia >30 mmol/L

  খ

  Ketonaemia < 3 mmol/L

  গ

  Serum bicarbonate >15 mmol/L

  ঘ

  Serum osmolarity >350 mosm/kg

  ঙ

  Acidosis ( PH <7.3 mmol/
• Symptoms of hyperglycaemia include
  ক

  Nausea

  খ

  Headache

  গ

  Apathy

  ঘ

  Weight gain

  ঙ

  Predilection for sour foods
• In oral glucose tolerance test
  ক

  8-10 hours needed for fasting

  খ

  Hyperglycaemia sine glycosuria can be interpreted

  গ

  Individual should be on normal diet for one day

  ঘ

  175 gm glucose in 250-300 ml water by mouth

  ঙ

  Renal threshold level within normal limit
• Hemoglobin Alc HbAlc level is used for
  ক

  Monitoring of glycemic control

  খ

  Diagnosis of diabetes mellitus

  গ

  Diagnosis of insulin resistance

  ঘ

  Diagnosis of thalassemia

  ঙ

  Diagnosis of sickle cell anemia