B. cirrhosis & liver failure MCQ 25+

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• In liver failure there is likely to be
  ক

  Salt & water deprivation

  খ

  Dependent oedema

  গ

  Impaired absorption of fat

  ঘ

  Intoxication after eating a high protein meal

  ঙ

  Raised blood urea
• Biochemical features of fulminating hepatic failure are
  ক

  Low blood urea nitrogen

  খ

  Prolongation of prothrombin time

  গ

  Low serum albumin

  ঘ

  Metabolic alkalosis

  ঙ

  Low blood ammonia level
• A cirrhosis case is likely to due to alpha 1 antitrypsin deficiency if
  ক

  The patient is pizz genotype

  খ

  If the patient shows typical clinical features at onset

  গ

  His/her liver biopsy shows alpha 1 antitrypsin globules

  ঘ

  His/her plasma alpha 1 antitrypsin is low

  ঙ

  He/she has early onset has severe disease
• Fulminant hepatitis is caused by
  ক

  Hepatitis A

  খ

  Hepatitis B

  গ

  Wilson disease

  ঘ

  Phenytoin

  ঙ

  Galactosaemia
• The following biochemical disturbances occur in fulminating hepatic failure
  ক

  Metabolic acidosis

  খ

  Hyperkalaemiaia

  গ

  Hypoglycaemia

  ঘ

  Fall in Po2

  ঙ

  Hypernatraemia
• Fulminant hepatic failure is characterized by
  ক

  Hepatic encephalopathy

  খ

  INR>3

  গ

  Coagulopathy

  ঘ

  Absence of hemorrhage

  ঙ

  Its occurrence usually 6 months after the onset of acute liver disease
• In cirrhosis of liver encephalopathy may be precipitated by
  ক

  Hypokalemia

  খ

  High protein diet

  গ

  Paracentesis

  ঘ

  Injection of vitamin K

  ঙ

  High carbohydrate diet
• Cirrhosis of liver is caused by the followings
  ক

  Hepatitis C virus

  খ

  Hepatitis B virus

  গ

  Hepatitis A virus

  ঘ

  Cytomegalovirus

  ঙ

  Epstein Barr virus
• In hepatic failure the following biochemical abnormalities may be found
  ক

  Increased plasma globulin

  খ

  Increased the blood ammonia concentration

  গ

  Impaired glucose tolerance

  ঘ

  Increased plasma albumin concentration

  ঙ

  Increased the blood urea
• Causes of chronic liver disease are
  ক

  Hepatitis D virus infection

  খ

  Hepatitis C virus infection

  গ

  Alpha 1 antitrypsin deficiency

  ঘ

  Hepatitis E virus infection

  ঙ

  Hepatitis A virus infection
• Following drugs are contraindicated in decompensated cirrhosis of liver
  ক

  Oral hypoglycemic agents

  খ

  Pegylated interferon

  গ

  Antihypertensives

  ঘ

  Propranolol

  ঙ

  Tenofovir
• Factors precipitating hepatic encephalopathy include
  ক

  Infection

  খ

  Constipation

  গ

  Hypokalaemia

  ঘ

  Antibiotics

  ঙ

  Excess dietary carbohydrate
• Cirrhotic ascites is characterized by
  ক

  Responsiveness to diuretics

  খ

  Raised glucose in ascitic fluid

  গ

  Low serum-ascitic albumin gradient

  ঘ

  More than 3.5 gm/dl total protein in ascitic fluid

  ঙ

  Milk white color of ascitic fluid
• Biochemical changes found in fulminant hepatic failure is
  ক

  Metabolic acidosis

  খ

  Prolongation of prothrombin time

  গ

  Hyperbilirubinemia

  ঘ

  Hyperglycemia

  ঙ

  Hypoalbuminemia
• Features of fulminant hepatic failure is/are
  ক

  Metabolic acidosis

  খ

  Prolonged prothrombin time

  গ

  Hyperbilirubinemia

  ঘ

  Hypercalcemia

  ঙ

  Low serum albumin
• Causes of cirrhosis of liver include
  ক

  Hepatitis C virus

  খ

  Hepatitis B virus

  গ

  Hepatitis E virus

  ঘ

  Cytomegalovirus

  ঙ

  Leptospirosis
• In acute liver failure
  ক

  Coagulopathy can be corrected by vitamin K

  খ

  Encephalopathy may occur

  গ

  Lactulose is beneficial

  ঘ

  Respiratory failure can occur

  ঙ

  Mannitol cannot be used
• Precipitating factors of hepatic encephalopathy includes
  ক

  Constipation

  খ

  Infection

  গ

  Portosystemic shunting

  ঘ

  Hyperkalaemia

  ঙ

  Volume overload
• Features of hepatocellular insufficiency include
  ক

  Jaundice

  খ

  Oesophageal varices

  গ

  Testicular atrophy

  ঘ

  Hypoalbuminemia

  ঙ

  Splenomegaly
• Features of hepatic decompensation include
  ক

  Hepatic encepahlopathy

  খ

  Jaundice

  গ

  Coagulopathy

  ঘ

  Ascites

  ঙ

  Gynaecomastia
• Feature/s of chronic liver disease is/are
  ক

  Palmar erythema

  খ

  Spider angioma

  গ

  Palpable purpura

  ঘ

  Granulma annulare

  ঙ

  Calcinosis cutis
• Features of decompensated liver disease are
  ক

  Ascites

  খ

  Coagulopathy

  গ

  Jaundice

  ঘ

  Spider angioma

  ঙ

  Testicular atrophy
• Feature/s of decompensated cirrhosis is/are
  ক

  Hyperbilirubinaemia

  খ

  Hypoalbuminaemia

  গ

  Hyperglycemia

  ঘ

  Prolongation of activated partial thromboplastin time (APT

  ঙ

  Prolongation of bleeding time